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Posted April 14, 2017: by Bill Sardi
Posted November 16, 2016: by Bill Sardi
The Statin Drug Shepherds Mislead The Sheep Again
Sheep have a strong instinct to follow the sheep in front of them. If one sheep in a large flock begins to bolt and run in a different direction the rest may follow. There is no question there is a statin-compliant patient who dutifully follows their doctor’s instructions without question and the rest follow. There is an overly loyal patient pool that follows the orders issue by their self-serving doctors.
Posted January 16, 2016: by Bill Sardi
I have written extensively at LewRockwell.com about cholesterol and statin drugs: A new injectable cholesterol drug (July 28, 2015); When will modern medicine give up its cholesterol cash-cow? (Dec 27, 2013); Statin cholesterol-lowering drugs work via reduction of iron (May 27, 2013); Statin drugs don’t save lives (Nov 10, 2008); Coronary calcium predicts future heart attacks, not cholesterol (March 28, 2008); Who will tell the people? It isn’t cholesterol. (Feb 5, 2007);
There have been numerous other critics of statin drugs: Don’t give more patients statins, John Abramson/Rita Redberg, NY times (Nov 13, 2013); The Great Cholesterol Con, Malcolm Kendrick (Oct 1, 2008); The Cholesterol Myths, Uffe Ravnskov (Oct 1, 2000); The Statin Damage Crisis, Duane Graveline (July 29, 2012); Lipitor Thief Of Memory, Duane Graveline (Sept 23, 2010)
Yet the American population has been systematically educated and can be considered a nation of ingrained cholesterol phobes and the statin drug industry continues to rake in billions of dollars in revenues.
Posted September 20, 2015: by Bill Sardi
Why do women experience a steep increase in heart attacks with the onset of menopause? The obvious answer, without checking the science, is the decline in estrogen production has something to do with the dramatic increase in strokes and heart attacks among post-menopausal women.
Despite the obvious benefits of estrogen replacement, the American Heart Association does not recommend postmenopausal hormone therapy to reduce the risk of coronary heart disease or stroke because some studies have failed to show a reduced risk. A spokesperson for the AHA says: “Estrogen decline isn’t the only reason women face a higher cardiovascular disease risk after reaching menopause. We’re trying to figure the rest of it out.” [American Heart Assn]
Posted September 9, 2015: by Bill Sardi
IP6 phytate is an overlooked nutritional factor that is protective against stiffening (calcification) of the first blood vessel (aorta) outside the heart. Stiffening of the aorta is associated with higher systolic (pumping) blood pressure.
The dietary intake of IP6 was recently measured as determined by excretion in urine among senior adults, with the following results:
Posted August 27, 2015: by Bill Sardi
Today’s news headlines once again attribute recent declines in coronary heart disease mortality to statin drugs and modern treatments like arterial stents.
Citing a report in the European Heart Journal, The Daily Mail, a British publication, mistakenly claims Great Britain has “one of the best records in Europe” with 184 deaths per 100,000 people for coronary heart disease — a dramatic 46.9% decline over the past 10 years. [European Heart Journal Aug 25, 2015; Daily Mail UK Aug 26, 2015] Yet the chart below reveals Great Britain (United Kingdom) isn’t even in the top 10 countries with the lowest death rate for coronary artery disease.
Posted July 27, 2015: by Bill Sardi
It’s a cardiologist’s dream come true. A newly approved $1200/monthly-injectable drug that dramatically reduces circulating cholesterol levels may become the first $100-200 billion medicine.
That would dwarf the record $9 billion annual sales for Lipitor, the statin cholesterol-lowering drug that was the best selling drug of all time until its patent recently expired. And to add value to the equation, this newly FDA-approved drug may be the first cholesterol-lowering medication to actually save lives, but for different reasons than advertised.
Posted April 15, 2015: by Bill Sardi
So you’ve had your heart attack or heart scare and cardiologists have relieved your unremitting chest pain by placement of wire props called stents in any of your four coronary arteries that supply the heart with oxygenated blood.
By now you’ve probably been placed on blood thinners and cholesterol-lowering drugs. But don’t fall into the trap of believing modern medicine’s false paradigm that cholesterol accumulation in your coronary arteries resulted in arterial narrowing and eventually a blood clot that caused your heart attack.
Before you become cholesterol-phobic it might be time to learn what really caused a blood clot to form in a coronary artery.
Posted April 4, 2015: by Bill Sardi
For a long time now I have listened to numerous heart attack victims, including a brother and four other male friends, explain symptoms that occurred when they experienced their crushing-chest-pain heart attack. In all these cases it sounded like they were having a gall bladder attack, not a heart attack.
Their stories were common: chest pain after a meal, strong acid reflux sometimes relieved by antacids and sometimes a history of prior gallstone problems or gall bladder removal.
Posted January 11, 2014: by Bill Sardi
Over a year ago I reported on a blood test that can predict an impending heart attack days prior to its occurrence. The test measured the number of circulating cells sloughed off from the inside of arteries that can block coronary arteries that supply oxygen to the heart. But I asked then, “what to do next?”
The test needed to be refined and validated, which is what Scripps Institute researchers announced recently in the journal of Physical Biology.
But precisely what would cardiologists do to prevent the onset of a heart attack if the test indicates a heart attack is imminent?
Most likely they will employ drugs that reduce coagulation (clotting) of the blood but the number of these microparticles must be reduced to address the problem directly.