Posted September 11, 2012: by Bill Sardi
Adult-onset diabetes is treated by a variety of drugs: an old standby, chlorpropamide (Diabinese), stimulates the pancreas to release more insulin. Newer drugs that perform the same function as Diabinese are exenatide (Byetta), glipizide (Glucotrol and Glucotrol XL), glyburide (Micronase, Glynase, and Diabeta), glimepiride (Amaryl), repaglinide (Prandin) and nateglinide (Starlix), but may not work any better than chlorpropamide. Then there are drugs that inhibit insulin resistance, such as Rosiglitazone (Avandia), pioglitazone (ACTOS), and metformin (Glucophage). Other anti-diabetic drugs like Acarbose (Precose) and meglitol (Glyset) help the body to lower blood sugar levels by blocking the breakdown of starches in the intestine. You would get the impression that diabetes is a drug deficiency rather than a diet-related/age-related disease.
Exactly what causes blood sugar levels to rise in middle-age? Researchers have known that answer to that question since 1994 – the accumulation of iron in the body. And it has been demonstrated numerous times that depletion of iron stores, as measured by the amount of an iron storage protein called ferritin, will produce long-term resolution of diabetes. Repeated blood donation can also accomplish this (a unit of blood contains about 250 milligrams of iron). Blood-letting combined with a natural iron chelator (key-lay-tor) like IP6 rice bran extract may eliminate the need to take drugs altogether. View the entire text of the recent report here.