Posted July 7, 2021: by Bill Sardi
If the public is to believe all of the unusual and atypical symptoms caused by COVID-19, uncharacteristic of any of the other seven types of coronaviruses, one might conclude this isn’t a virus at all. It must be something else. In particular, modern medicine is perplexed over patients with long-term symptomology, what has now been called long-haul COVID-19.
Researchers at the Max Planck Institute For Physical Medicine in Germany find red blood cells from recovered long-haul COVID-19 patients to be misshapen which they say may explain the phenomenon of oxygen deprivation and other symptoms among these patients.
Writing in The Biophysical Journal, investigators assessed 4 million red blood cells from healthy, infected and recovering COVID-19 patients. Actively-infected patients have larger red blood cells, and 7 months after hospitalization their red blood cells are smaller (see graphic).
Investigators say these deformed cells could explain the increased risk for blood clots or embolisms (blood clot released to another organ like the lungs or brain).
According to one report, both COVID-19 infection and vaccination increase the risk for blood clots, though in hard numbers the risk is small, a few in a million. Active infection increases the risk 8-10 times more than vaccination however. Clots were 100 times more common among COVID-19 infected patients than healthy adults. But long-term data isn’t available regarding the risk for blood clots upon reinfection with a coronavirus.
Researchers at the Michigan Medicine Frankel Cardiovascular Center believe this COVID-19 blood clotting problem emanates from antiphospholipid antibodies. Surprisingly, half of hospitalized COVID-19 patients were positive for these auto-antibodies. Furthermore, these patients also had super-activated neutrophils, a type of white blood cell that can potentially be destructive if overactivated. These investigators say there are clueless as to what is causing this.
Meanwhile, researchers reporting in the CRITICAL CARE Journal may have an answer to this perplexing problem. They employed 200 milligrams of daily vitamin B1 (thiamine) intravenously and achieved a 75% reduction in absolute risk for mortality among hospitalized COVID-19 patients compared to non-B1-treated patient. Additionally, B1 therapy reduced risk for thrombosis (blood clots) by 81%!
Thiamin B is not known for having anti-blood clotting properties. It does not make blood platelets less sticky and prone to clot.
However, the lack of thiamine may induce megaloblastic anemia where there are fewer red blood cells and remaining cells are larger than normal.
Severe cases of COVID-19 are associated with a decline in blood platelets which clump to form blood clots. The use of blood-thinning drugs may be life-saving but the cause of this blood coagulation disorder may not be properly addressed.
In COVID-19 blood platelets may be abnormally activated, leading to hyper-clotting.
Thiamine B1 deficiency is reported to induce thrombocytopenia – – low blood platelet count. (Platelets facilitate blood clots). Low blood platelets are linked to a vitamin B1 deficiency.
Women with COVID-19 report cases of abnormal menstrual bleeding. So far, the reason for this is unknown. But we do know that the provision of thiamine B1 is reported to reduce abnormal menstrual bleeding.
In cases of COVID-19, patients may suffer from dangerously low oxygen levels even though they do not exhibit signs of lung abnormalities. They may not have shortness of breath but they may exhibit “silent hypoxia,” unexplained air hunger.
COVID-19 patients are often reported to have faintly blue lips and fingertips. This has baffled doctors. It resembles high-altitude sickness. Dr. Derrick Lonsdale, author of books about vitamin B1, says this is a characteristic sign of beriberi – vitamin B1 deficiency.
In living tissues, the lack of oxygen (hypoxia) increases thiamine transporters by 31-fold and a 2-fold net increased delivery of oxygen to tissues. The lack of thiamine impairs the production of nitric oxide, a transient intra-arterial gas that dilates (widens) arteries to facilitate delivery of oxygen-carrying red blood cells and regulate blood pressure. A shortage of thiamin B1 promotes a protein called hypoxia inducing factor.
An underlying environmental factor may set up human populations for oxygen starvation by COVID-19 patients. The herbicide Roundup/glyphosate (Monsanto) contains arsenic that can set the stage of oxygen starvation diseases. Arsenic is a toxic mineral that is widely found in water and other environmental sources and may induce pseudo-hypoxia.
This author has attempted to alert doctors that the symptoms of COVID-19, particularly the long-haul cases, and the cases where there are positive blood tests but no symptomology, may be a stealth vitamin B1 deficiency. I have written seven reports linking COVID-19 to a vitamin B1 deficiency. This is the eighth. I’ve sent countless reports to physicians at leading medical institutions.
So far, there has been almost no acknowledgement from physicians that COVID-19 is really a vitamin deficiency induced by anxiety and fear that increased consumption of alcohol, coffee and tea that contain anti-vitamin B1 factors.
Even when I contacted a major manufacturer of thiamin B1 in Japan to educate them about this link to COVID-19, they refused to talk with me, as if they know there will be negative consequences for telling the world about this.
We have two coincident pandemics underway; one contrived by recategorizing colds (coronavirus infections), or cases of influenza, or even tuberculosis, as COVID19; and the other a real pandemic as Americans drank 500% more alcohol and increased consumption of coffee and tea, all three beverages interfering with vitamin B1 metabolism. Obviously, the vitamin B1 explanation doesn’t fit narratives that falsely claim vaccines are the antidote to all infections, viral or otherwise.
And if many cases of COVID-19 are not caused by viruses but rather an unwitting self-induced vitamin deficiency, then precisely what are the vaccines, other than bioweapons?
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