Posted April 15, 2015: by Bill Sardi
So you’ve had your heart attack or heart scare and cardiologists have relieved your unremitting chest pain by placement of wire props called stents in any of your four coronary arteries that supply the heart with oxygenated blood.
By now you’ve probably been placed on blood thinners and cholesterol-lowering drugs. But don’t fall into the trap of believing modern medicine’s false paradigm that cholesterol accumulation in your coronary arteries resulted in arterial narrowing and eventually a blood clot that caused your heart attack.
Before you become cholesterol-phobic it might be time to learn what really caused a blood clot to form in a coronary artery.
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Posted April 4, 2015: by Bill Sardi
For a long time now I have listened to numerous heart attack victims, including a brother and four other male friends, explain symptoms that occurred when they experienced their crushing-chest-pain heart attack. In all these cases it sounded like they were having a gall bladder attack, not a heart attack.
Their stories were common: chest pain after a meal, strong acid reflux sometimes relieved by antacids and sometimes a history of prior gallstone problems or gall bladder removal.
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Posted January 11, 2014: by Bill Sardi
Over a year ago I reported on a blood test that can predict an impending heart attack days prior to its occurrence. The test measured the number of circulating cells sloughed off from the inside of arteries that can block coronary arteries that supply oxygen to the heart. But I asked then, “what to do next?”
The test needed to be refined and validated, which is what Scripps Institute researchers announced recently in the journal of Physical Biology.
But precisely what would cardiologists do to prevent the onset of a heart attack if the test indicates a heart attack is imminent?
Most likely they will employ drugs that reduce coagulation (clotting) of the blood but the number of these microparticles must be reduced to address the problem directly.
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Posted November 13, 2013: by Bill Sardi
Nov. 12, 2013- Out with monitoring cholesterol numbers. In what is being called a “tectonic shift in the way doctors treat high cholesterol,” the American Heart Association unexpectedly announced new guidelines today for prescribing statins drugs.
Without admission that older guidelines were a misdirection, the new guidelines abandon the idea of lowering “bad” LDL cholesterol to below 100 and re-set that number at 190. No mention is made about raising HDL “good” cholesterol, which cardiologists also once embraced till a major study had to be halted when elevation of HDL cholesterol resulted in increased deaths.
Cardiologists hope to get it right this time saying subjects who don’t fit the newly established guidelines should be encouraged to implement lifestyle changes such as smoking cessation along with dietary and exercise regimens.
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Posted August 16, 2013: by Bill Sardi
In the looming era of the Affordable Care Act, where all medical technologies must be validated scientifically or billings for medicines, devices or services will not be fully reimbursed, modern medicine is struggling to substantiate its many therapies.
An example is a recent study published in The American Journal of Medicine. Researchers attempted to blame the inability to demonstrate reduction in cardiovascular death over a period of 4 years on poor patient compliance.
Less than half of these high-risk patients (48.2%) fully adhered to medication regimens.
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Posted August 15, 2013: by Bill Sardi
RADIO BROADCAST KLAV-1230AM, Las Vegas, NV, August 15,2013
Dear Doctor:
If there is any doctor listening out there…
I read your best medical journals, as I’m sure you do.
I know the difference between statistical and clinical improvements.
I know that the central focus of modern medicine in the past four decades, cholesterol reduction with statin drugs, has not significantly lowered mortality among high-risk or healthy adults (if you don’t believe what I just said, then check the bulletin just issued in the Journal of the American Medical Association). Harvard Dr. John Abramson, author of Overdosed America, has also documented that fact among healthy adults. The alarming part of the newly issued AMA bulletin is that the assumed benefits of lowering mortality among high-risk statin drug users has also been a false assumption.
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Posted August 12, 2013: by Bill Sardi
Comment: The newly released report (click link to review), published in the Journal of the American Medical Association (JAMA), concedes that statin drugs have now been relegated as secondary intervention in the prevention of heart disease. What now serves as primary prevention in its place, according to the JAMA report, are control of “lifestyle factors” such as smoking cessation, dietary measures, and limitation of alcohol intake).
This is a nice way of saying the major direction of western medicine in the last forty years has been flawed, even corrupt. Unfortunately, the only penalty for being wrong has been experienced by those who took statin drugs in good faith, paid the price with their lives, believing their well-trained physicians were guiding them in the right direction. That wasn’t true, and cardiologists knew this for a long time prior to the published report below.
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Posted August 5, 2013: by Bill Sardi
Something huge is going on in the cholesterol world. Those lipoproteins we have been taught to be phobic over – LDL (low density “bad” cholesterol), total cholesterol and triglycerides, as well as HDL cholesterol (high density “good” lipoproteins), believed to clog arteries and induce heart attacks, are losing scientific ground as true measures of your risk for a mortal heart attack. Another lipoprotein that was cast aside decades ago is gaining attention.
That lipoprotein is called lipoprotein(a). Readers may recall it is the cholesterol particle that was temporarily made famous by vitamin C researchers Drs. Linus Pauling and Matthias. Since their research in the 1990s lipoprotein(a) has been largely shunned and ignored.
Before I go on to write about lipoprotein(a), I am forced to address the shortcomings of the cholesterol theory of heart disease. It takes a strong amount of evidence to convince anyone, especially gullible statin drug users, that cholesterol is a mistaken direction in modern medicine.
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Posted July 12, 2013: by Bill Sardi
It is strange that modern medicine has no arterial cleansing regimens beyond that of ineffective statin cholesterol-lowering drugs. Statin drugs only reduce production of cholesterol from the liver, thus reducing circulating levels of cholesterol, not cholesterol plaque itself.
The unnatural and problematic method by which statin drugs lower cholesterol has been graphically portrayed. Cholesterol numbers are essentially worthless in gauging whether a person is at risk for a sudden mortal heart attack.
More troubling is the realization that baby-dose (81 milligram) aspirin tablets with a red heart on the bottle to signify they promote heart health, do not protect against sudden mortal heart attacks. An estimated 40 million Americans take a baby aspirin in an ineffective attempt to reduce their risk of dying suddenly from a fast-forming blood clot in a coronary artery that supplies the heart with oxygenated blood.
The realization that aspirin and statin drugs are ineffective and even problematic prompts a search for other ways to maintain arterial health.
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Posted May 26, 2013: by Bill Sardi
Every once in a while some brave investigator charts a new course for modern medicine. For various reasons this one is not likely to become the new paradigm and supplant the reigning cholesterol theory of heart disease. But a recently published landmark paper posits a long overlooked hypothesis which gleans evidence from statin cholesterol-lowering drugs themselves. It convincingly points those health practitioners who embrace science over commercial gain in a direction away from cholesterol as a measure of arterial health.
Leo R Zacharski MD, a medical and scientific board member of The Iron Disorders Institute, writes a seminal paper entitled “THE STATIN-IRON NEXUS: ANTI-INFLAMMATORY INTERVENTION FOR ARTERIAL DISEASE PREVENTION in the April issue of The American Journal of Public Health.
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